Biological risks for neurological abnormalities associated with hyperbilirubinemia
Unconjugated bilirubin (UCB) injury to glial cells leads to the secretion of glutamate and elicits a typical inflammatory response. Release of pro-inflammatory cytokines may influence gliogenesis and neurogenesis, and lead to deficits in learning and memo. - Fundacao para a Ciencia e a Tecnologia (F...
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Other Authors: | , , , , |
Format: | article |
Language: | eng |
Published: |
2015
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Subjects: | |
Online Access: | http://hdl.handle.net/10451/21538 |
Country: | Portugal |
Oai: | oai:repositorio.ul.pt:10451/21538 |