TrkB-ICD fragment, originating from BDNF receptor cleavage, is translocated to cell nucleus and phosphorylates nuclear and axonal proteins

Copyright © 2019 Fonseca-Gomes, Jerónimo-Santos, Lesnikova, Casarotto, Castrén, Sebastião and Diógenes. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the ori...

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Detalhes bibliográficos
Autor principal: Fonseca-Gomes, João (author)
Outros Autores: Jerónimo-Santos, André (author), Lesnikova, Angelina (author), Casarotto, Plinio (author), Castrén, Eero (author), Sebastião, Ana M (author), Diógenes, Maria José (author)
Formato: article
Idioma:eng
Publicado em: 2022
Assuntos:
Alzheimer’s disease
TrkB-FL cleavage
TrkB-ICD fragment
Brain-derived neurotrophic factor (BDNF)
Excitotoxicity
Neurodegeneration
Neuroprotection
Texto completo:http://hdl.handle.net/10451/54165
País:Portugal
Oai:oai:repositorio.ul.pt:10451/54165
Descrição
Resumo:Copyright © 2019 Fonseca-Gomes, Jerónimo-Santos, Lesnikova, Casarotto, Castrén, Sebastião and Diógenes. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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