Inhibition of inducible nitric oxide synthase reduces renal ischemia/reperfusion injury

Background. Nitric oxide (NO), produced via inducible nitric oxide synthase (iNOS), is implicated in the pathophysiology of renal ischemia/reperfusion (I/R) injury. The aim of this study was to investigate the effects of the iNOS inhibitors L-N-6-(1-iminoethyl)lysine (L-NIL) and aminoethyl-isothiour...

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Bibliographic Details
Main Author: Chatterjee, PK (author)
Other Authors: Patel, NSA (author), Kvale, EO (author), Cuzzocrea, S (author), Brown, PAJ (author), Stewart, KN (author), Mota-Filipe, H (author), Thiemermann, C (author)
Format: article
Language:eng
Published: 2015
Subjects:
Urology & Nephrology
Online Access:http://hdl.handle.net/10451/21216
Country:Portugal
Oai:oai:repositorio.ul.pt:10451/21216

Internet

http://hdl.handle.net/10451/21216

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